Uric acid
CAS No. : 69-93-2
| Size | Price | Stock |
|---|---|---|
| 5g | $27 | In-stock |
| 25g | $59 | In-stock |
| 100g | $126 | In-stock |
| 500g | $618 | In-stock |
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| Cat. No. : | HY-B2130 |
| M.Wt: | 168.11 |
| Formula: | C5H4N4O3 |
| Purity: | >98 % |
| Solubility: | H2O : 6.25 mg/mL (ultrasonic;adjust pH to 10 with 1M NaOH);DMSO : 0.67 mg/mL (ultrasonic);1M NaOH : 8.33 mg/mL (ultrasonic;warming;adjust pH to 12 with 1M NaOH;heat to 60°C) |
Uric acid, scavenger of oxygen radical, is a very important antioxidant that help maintains the stability of blood pressure and antioxidant stress. Uric acid can remove reactive oxygen species (ROS) such as singlet oxygen and peroxynitrite, inhibiting lipid peroxidation[1][2].
In Vitro:Uric acid (400 μM; 48 hours) protects Caco-2 cells from indomethacin-induced lipid peroxidation[2].
Co-treatment of cells with Indomethacin and Uric acid (200 μM IND plus 400 μM UA; 24 hours) significantly decreases ROS levels compared to those in cells incubated with indomethacin alone. Cell viability in Caco-2 cells treated with both Indomethacin and Uric acid (200 μM IND plus 400 μM UA; 24 hours) is higher than that in cells treated with indomethacin alone. Uric acid has a protective effect on indomethacin-induced intestinal cell changes through its antioxidant activity[2].
In Vivo:Uric acid (250 mg/kg; p.o.) ameliorates indomethacin-induced enteropathy[2].
Oral administration of uric acid decreased ROS accumulation in the ileum in a mouse model of indomethacin-induced enteropathy[2].
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